| 1. | Graft survival , but not patient survival , was influenced by the presence of nas
Nas对移植肝存活有影响，而对患者存活无影响。 |
| 2. | Hepatic venous complications were rare . patient and graft survival were not different between cca and control patients
肝静脉的并发症很少。在病人和移植肝的生存期方面cca组和对照组没有差异。 |
| 3. | With administration of ctla4 - ig , short - term blockade of cd28 - mediated corecognition resulted in prolonged graft survival and tolerance in mhc mismatched rodent models
给予huctla4 - ig ，短期阻断cd28介导的共识别可导致mhc不匹配的啮齿类模型中移植物的长期存活与耐受。 |
| 4. | Durable immune tolerance supporting vascularized allotransplantation offers the possibility of extending graft survival and avoiding harmful complications of chronic immunosuppression
血管化的异体移植的持久的免疫耐受能够提高移植物的存活率，并避免慢性免疫抑制带来的不良并发症。 |
| 5. | The incidence of acute rejection was similar between the two groups whereas the initial graft survival advantage in favor of the hb group diminished gradually over the course of time
两组的急性排斥反应发生率无明显差异，而hb组在移植肾早期存活率方面的优势将随着时间推移而逐渐消失。 |
| 6. | Liver transplantation with neoadjuvant therapy is associated with far higher rates of late arterial and portal venous complications , but these complications do not adversely affect patient and graft survival
新辅助治疗后的肝移植伴发很高的迟发性动脉和门静脉并发症，但这些并发症并不会对病人和移植肝的存活期产生负面影响。 |
| 7. | The following outcomes were evaluated : warm and cold ischemia times , primary nonfunction , delayed graft function , length of hospital stay , acute graft rejection , patient and graft survival , and post - transplant serum creatinine
对以下指标进行了评估：热缺血和冷缺血时间、原发性无功能、移植肾功能延迟恢复、住院时间、急性排斥反应、病人及移植肾存活时间、移植后血肌酐水平。 |
| 8. | On the other hand , there is substanial evidences tha dcs mediate tolerane . the gm - csf - stbolated mouse bone mwderived mhc class ii + dc progendors tha are deficient in cell surface erpression of the costbolatory molecules b7 - l and b7 - 2 can induce alloanginspecific t ceil anergy in vbo . systendc adchstraion of these donor - derived dc progendors to recipients intravenously 7 days before transplantation prolonged the median graft survival tane from 9 . 5 days to 22 days wbout additional twnosuppressions in a mouse cardinc twlanation model
1995年， lul等报道表面缺乏共刺激分子，特别是缺乏cd80 ( b7 - 1 )和cd86 ( b7 - 2 )的未成熟型dc能在体外诱导同种抗原特异性的t细胞无能，而对dc的适当处理能够使未成熟型dc潜在的耐受原性得以放大和增强，使得dc在诱导免疫耐受中具有相当重要的作用及潜在的应用价值。 |
