| 1. | Inhibition of ros activity by catalase , n - acetylcysteine or diphenylene iodonium abrogated low k + - induced increases in the - subunit promoter activity , 1 - and 1 - subunit polypeptide abundance , and binding sites of na , k - atpase , indicating the role of ros in the effect of low k +
抗氧化剂nac与catalase 、 dpi能抑制低钾的作用。提示ros产生与细胞膜nadph氧化酶有关。进一步研究表明， h2o2 ，而非超氧根离子参与了低钾对钠钾atp酶的调节。 |
