| 1. | Involvement of pressure - related mechanism in activation of inducible nitric oxide synthase
诱导型一氧化氮合酶的激活与血压的关系 |
| 2. | The activity of inducible nitric oxide synthase ( inos ) was determined using inos assay kit
用诱导型一氧化氮合酶( inos )检测试剂盒测定胞质内inos的活性。 |
| 3. | Effect of niupozhibao weiwan on cerebral inducible nitric oxide synthase expression of rats with endotoxic shock
牛珀至宝微丸对内毒素休克大鼠脑诱导型一氧化氮合酶表达的影响 |
| 4. | Effect of extract of ginkgo biloba leaves on expression of inducible nitric oxide synthase after sciatic nerve injury : experiment with rats
银杏酮酯对大鼠坐骨神经损伤后诱导型一氧化氮合酶表达的影响 |
| 5. | Influence of cryopreservation time on the expression of graft inducible nitric oxide synthase after orthotopic liver transplantation in rats
冷保存时间对大鼠原位肝移植后移植物诱导型一氧化氮合酶表达的影响 |
| 6. | Expressions of heme oxygenase 1 and inducible nitricoxide synthase in renal tissues of rats with intestinal ischemia - reperfusion injury
血红素加氧酶1与诱导型一氧化氮合酶在肠缺血再灌注损伤大鼠肾组织中的表达 |
| 7. | Effects of basic fibroblast growth factor on proliferation of human lens epithelial cells , nitric oxide and inducible nitric oxide synthase
碱性成纤维细胞生长因子对人晶体上皮细胞增殖及一氧化氮和诱导型一氧化氮合酶的影响 |
| 8. | This article reviews the development on studies of the structure , function and catalytic pathway of nitric oxide synthase ( nos ) . the structure - function relationship and oxygenation mechanism of the inos were discussed
摘要综述了一氧化氮合酶的结构、功能和催化路径，讨论了诱导型一氧化氮合酶的结构和功能的关系以及加氧氧化的机理。 |
| 9. | No , a first gas information molecule discovered in human being , is a typical endothelial - derived relaxant and mediates endothelium - dependent relaxation of blood vessels . in the pathogesis of endotoxin shock vec is one of the major target cells of lps and lps - induced proinflammatory cytokine such as tumor necrosis factor and interlukin 1 and activated . in vec inducible nitric oxide synthase ( inos ) is induced and lead to an increase in production of no , the while endothelial nitric oxide synthase ( enos ) is inhibited and elicit decrease in no formation , both of which are demonstrated to induce the
在内毒素休克过程中vec是lps及其诱导机体产生的多种促炎细胞因子如tnf 、 il - 1作用的主要靶细胞, vec诱导型一氧化氮合酶( induciblenitricoxidesynthase , inos )激活、 no大量诱生而内皮型一氧化氮合酶( endothelialnitricoxidesynthase , enos )活性被抑制、 no生成障碍,是血管反应性异常变化、血管调节机制紊乱的重要发病环节。 |
