| 1. | To explore the role of ccr5 as a marker for monitoring hiv infection
研究趋化因子受体第5号在监察爱滋病病毒感染方面的标志作用。 |
| 2. | To determine the functional and or quantitative deficiency in ccr5 expressing cells ; and
确定趋化因子受体第5号的表现细胞在功能上及或数量上的不足以及 |
| 3. | To determine the functional andor quantitative deficiency in ccr5 expressing cells ; and
确定趋化因子受体第5号的表现细胞在功能上及或数量上的不足；以及 |
| 4. | To mark out the pattern of ccr5 expression on antigen - presenting cells apcs in hiv infection
鉴定趋化因子受体第5号在感染爱滋病病毒者的呈抗原细胞上的表现模式 |
| 5. | To mark out the pattern of ccr5 expression on antigen - presenting cells ( apcs ) in hiv infection
鉴定趋化因子受体第5号在感染爱滋病病毒者的呈抗原细胞上的表现模式； |
| 6. | The expression of erythrocyte chemokine receptor on peripheral blood red blood cells of patients with psoriasis
红细胞趋化因子受体在寻常型银屑病患者外周血红细胞中的表达及其临床意义 |
| 7. | To determine the prevalence of the major forms of ccr5 promotor and ccr2 polymorphism in healthy chinese adults ; and
确定在健康的成年华人身上出现主要形式的趋化因子受体第5号催化剂和趋化因子受体第2号变异的比率以及 |
| 8. | To determine the prevalence of the major forms of ccr5 promotor and ccr2 polymorphism in healthy chinese adults ; and
确定在健康的成年华人身上出现主要形式的趋化因子受体第5号催化剂和趋化因子受体第2号变异的比率；以及 |
| 9. | Chemotaxis mediated by chemokine receptors , such as cxcr4 , play a key role in lymphocyte homing and hematopoiesis as well as in breast cancer metastasis . we have previously demonstrated that ? - arrestin2 functions to attenuate cxcr4 - meidated g protein activation and to enhance cxcr4 internalization . here we further show that expression of ( - arrestin2 in both hela and hek293 cells significantly enhanced the chemotactic efficacy of stromal - cell derived factor 1 ( ( sdf - 1 ( ) , the specific agonist of cxcr4
- arrestin2是趋化因子受体的一种重要的调节蛋白，本研究工作发现在hek - 293或hela细胞中升高- arrestin2的表达水平会显著增强cxcr4介导的趋化作用，反之当- arrestin2的表达被它的反义核苷酸或rnai所抑制， cxcr4介导的趋化作用则被明显抑制。 |
| 10. | While the suppression of ( - arrestin2 endogenous expression by antisense or rnai technology considerably attenuated sdf - 1 ( - induced cell migration . expression of ( - arrestin2 also augmented chemokine receptor ccr5 - mediated but not egf receptor - mediated chemotaxis , indicating the specific effect of ( - arrestin2
与此相似，另一种趋化因子受体ccr5介导的趋化作用也受- arrestin2的调控，但egf受体介导的趋化作用则不受- arrestin2的调控，说明- arrestin2的作用有一定的受体特异性。 |
